Human SLE variant NCF1-R90H promotes kidney damage and murine lupus through enhanced Tfh2 responses induced by defective efferocytosis of macrophages

Overview of attention for article published in Annals of the Rheumatic Diseases, September 2021

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Mice with a hypofunctional Ncf1 gene variant develop spontaneous #autoimmunity with high #interferon levels and exacerbated #glomerulonephritis after pristane treatment https://t.co/5zT7cWfp60

PC | Human SLE variant NCF1-R90H promotes kidney damage and murine lupus through enhanced Tfh2 responses induced by defective efferocytosis of macrophages https://t.co/FCwxCIqQof

GC | Human SLE variant NCF1-R90H promotes kidney damage and murine lupus through enhanced Tfh2 responses induced by defective efferocytosis of macrophages https://t.co/2Zh0ara9PJ

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