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Changes in alpha-SMA alter both the conformation of focal adhesions and the constitutive activity levels of FAK. http://t.co/4wxXn0P7Lj
Smooth muscle hyperplasia due to loss of smooth muscle α-actin is driven by activation of focal adhesion kinase, altered p53 localization and increased levels of platelet-derived growth factor…
Overview of attention for article published in Human Molecular Genetics, April 2013