Transient mitochondrial DNA double strand breaks in mice cause accelerated aging phenotypes in a ROS-dependent but p53/p21-independent manner

Overview of attention for article published in Cell Death & Differentiation, December 2016

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In the top 25% of all research outputs scored by Altmetric
High Attention Score compared to outputs of the same age (90th percentile)
High Attention Score compared to outputs of the same age and source (89th percentile)

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From: Cell Death Differentiation Transient mitochondrial DNA double strand breaks in mice cause accelerated ... https://t.co/9egW1QiW7D

From: Cell Death Differentiation Transient mitochondrial DNA double strand breaks in mice cause accelerated aging phenotypes in a ROS-dependent but p53/p21-independent manner https://t.co/9egW1QiW7D

Transient mitochondrial DNA double strand breaks in mice cause accelerated aging phenotypes in a ROS-dependent but p53/p21-independent manner. https://t.co/wj52fmo7vu #Neuralstemcells

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