| Title |
BAS1: A gene regulating brassinosteroid levels and light responsiveness in Arabidopsis
|
|---|---|
| Published in |
Proceedings of the National Academy of Sciences of the United States of America, December 1999
|
| DOI | 10.1073/pnas.96.26.15316 |
| Pubmed ID | |
| Authors | |
| Abstract |
The Arabidopsis bas1-D mutation suppresses the long hypocotyl phenotype caused by mutations in the photoreceptor phytochrome B (phyB). The adult phenotype of bas1-D phyB-4 double mutants mimics that of brassinosteroid biosynthetic and response mutants. bas1-D phyB-4 has reduced levels of brassinosteroids and accumulates 26-hydroxybrassinolide in feeding experiments. The basis for the mutant phenotype is the enhanced expression of a cytochrome P450 (CYP72B1). bas1-D suppresses a phyB-null allele, but not a phyA-null mutation, and partially suppresses a cryptochrome-null mutation. Seedlings with reduced BAS1 expression are hyperresponsive to brassinosteroids in a light-dependent manner and display reduced sensitivity to light under a variety of conditions. Thus, BAS1 represents one of the control points between multiple photoreceptor systems and brassinosteroid signal transduction. |
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Mendeley demographics
Geographical breakdown
| Country | Count | As % |
|---|---|---|
| United States | 2 | 1% |
| Hungary | 1 | <1% |
| France | 1 | <1% |
| Germany | 1 | <1% |
| China | 1 | <1% |
| Belgium | 1 | <1% |
| Unknown | 170 | 96% |
Demographic breakdown
| Readers by professional status | Count | As % |
|---|---|---|
| Student > Ph. D. Student | 40 | 23% |
| Researcher | 38 | 21% |
| Student > Master | 15 | 8% |
| Professor | 13 | 7% |
| Professor > Associate Professor | 10 | 6% |
| Other | 29 | 16% |
| Unknown | 32 | 18% |
| Readers by discipline | Count | As % |
|---|---|---|
| Agricultural and Biological Sciences | 104 | 59% |
| Biochemistry, Genetics and Molecular Biology | 20 | 11% |
| Chemistry | 4 | 2% |
| Computer Science | 2 | 1% |
| Medicine and Dentistry | 2 | 1% |
| Other | 6 | 3% |
| Unknown | 39 | 22% |