Innate Lymphoid Cells Are the Predominant Source of IL-17A during the Early Pathogenesis of Acute Respiratory Distress Syndrome

Overview of attention for article published in American Journal of Respiratory & Critical Care Medicine, February 2016

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@ErinMcCreary 1/ IL-6 prob contributes some to lung inflammation via stimulating Th17s to produce IL-17A, but targeting IL-17A directly makes more sense #inmice NLRP3 induced ARDS, primary source IL-17A are lung resident (NCR-) ILCs. pILC3 >> Tc17…

3/ #inmice LPS-induced (NLRP3) ARDS model, the neutrophilic inflammation is mainly attributed to IL-17A produced by tissue resident ILC2 cells. https://t.co/l1pbMzy1tI

@J_Immunol @pandoralucrezia @DocMMW In NLRP3 ARDS model #inmice most IL-17A secreted from lung resident ILC2s (NCR-/cKit+ designated "pILC3s" in paper), then Tc17~Th17>(γδ) In CoV2, tissues w/inflammation/hypoxia (even distal 2 infection sites), all …

RT @JukeBaRosh: 2/ ILC2s are plastic & can switch b/w pro or anti-inflammatory forms. Inflammatory ILC2s are the major source of IL-17A in…

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