Mechanisms of SARS-CoV-2 Infection-Induced Kidney Injury: A Literature Review

@go_howl @ChildrensHD https://t.co/RW48pBYZyv

https://t.co/hZ2EWiyYgT "These factors contribute to the AKI in #COVID19 cases by promoting tubular and endothelial dysfunction." "Dealing with RAS imbalances will be the key to the problem."

https://t.co/hZ2EWiyYgT "It also stimulates the production of cytokines/chemokines to cause immune cells (neutrophils, mononuclear cells, T cells, and B cells) to infiltrate the injury site and enhance the inflammatory response."

https://t.co/hZ2EWiyYgT "Furthermore, Ang II interacts with kidney resident cells to promote the production of pro-inflammatory factors, including prostaglandins, vascular endial cell growth factor, nuclear factor kappa B, TNFα, IL-1β, IL-6, and IFN-γ."