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Mendeley readers
| Title |
Abnormal interneuron development in disrupted-in-schizophrenia-1 L100P mutant mice
|
|---|---|
| Published in |
Molecular Brain, April 2013
|
| DOI | 10.1186/1756-6606-6-20 |
| Pubmed ID | |
| Authors |
Frankie HF Lee, Clement C Zai, Sabine P Cordes, John C Roder, Albert HC Wong |
| Abstract |
Interneuron deficits are one of the most consistent findings in post-mortem studies of schizophrenia patients and are likely important in the cognitive deficits associated with schizophrenia. Disrupted-in-Schizophrenia 1 (DISC1), a strong susceptibility gene for schizophrenia and other mental illnesses, is involved in neurodevelopment, including that of interneurons. However, the mechanism by which DISC1 regulates interneuron development remains unknown. In this study, we analyzed interneuron histology in the Disc1-L100P single point mutation mouse, that was previously shown to have behavioral abnormalities and cortical developmental defects related to schizophrenia. |
Mendeley readers
The data shown below were compiled from readership statistics for 82 Mendeley readers of this research output. Click here to see the associated Mendeley record.
Geographical breakdown
| Country | Count | As % |
|---|---|---|
| Spain | 1 | 1% |
| United States | 1 | 1% |
| China | 1 | 1% |
| Brazil | 1 | 1% |
| Unknown | 78 | 95% |
Demographic breakdown
| Readers by professional status | Count | As % |
|---|---|---|
| Student > Ph. D. Student | 27 | 33% |
| Student > Master | 11 | 13% |
| Student > Doctoral Student | 9 | 11% |
| Student > Bachelor | 8 | 10% |
| Researcher | 4 | 5% |
| Other | 13 | 16% |
| Unknown | 10 | 12% |
| Readers by discipline | Count | As % |
|---|---|---|
| Agricultural and Biological Sciences | 30 | 37% |
| Neuroscience | 16 | 20% |
| Medicine and Dentistry | 8 | 10% |
| Biochemistry, Genetics and Molecular Biology | 7 | 9% |
| Psychology | 5 | 6% |
| Other | 2 | 2% |
| Unknown | 14 | 17% |