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BAK overexpression mediates p53-independent apoptosis inducing effects on human gastric cancer cells

Overview of attention for article published in BMC Cancer, July 2004
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About this Attention Score

  • Good Attention Score compared to outputs of the same age (66th percentile)
  • Good Attention Score compared to outputs of the same age and source (72nd percentile)

Mentioned by

wikipedia
1 Wikipedia page

Citations

dimensions_citation
27 Dimensions

Readers on

mendeley
13 Mendeley
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Title
BAK overexpression mediates p53-independent apoptosis inducing effects on human gastric cancer cells
Published in
BMC Cancer, July 2004
DOI 10.1186/1471-2407-4-33
Pubmed ID
Authors

Qiang-Song Tong, Li-Duan Zheng, Liang Wang, Jun Liu, Wei Qian

Abstract

BAK (Bcl-2 homologous antagonist/killer) is a novel pro-apoptotic gene of the Bcl-2 family. It has been reported that gastric tumors have reduced BAK levels when compared with the normal mucosa. Moreover, mutations of the BAK gene have been identified in human gastrointestinal cancers, suggesting that a perturbation of BAK-mediated apoptosis may contribute to the pathogenesis of gastric cancer. In this study, we explored the therapeutic effects of gene transfer mediated elevations in BAK expression on human gastric cancer cells in vitro.

Mendeley readers

The data shown below were compiled from readership statistics for 13 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Indonesia 1 8%
Unknown 12 92%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 7 54%
Student > Master 2 15%
Student > Bachelor 2 15%
Student > Postgraduate 1 8%
Researcher 1 8%
Other 0 0%
Readers by discipline Count As %
Agricultural and Biological Sciences 6 46%
Biochemistry, Genetics and Molecular Biology 3 23%
Medicine and Dentistry 3 23%
Materials Science 1 8%

Attention Score in Context

This research output has an Altmetric Attention Score of 3. This is our high-level measure of the quality and quantity of online attention that it has received. This Attention Score, as well as the ranking and number of research outputs shown below, was calculated when the research output was last mentioned on 19 December 2006.
All research outputs
#3,499,367
of 12,222,940 outputs
Outputs from BMC Cancer
#952
of 4,482 outputs
Outputs of similar age
#80,519
of 270,334 outputs
Outputs of similar age from BMC Cancer
#19
of 76 outputs
Altmetric has tracked 12,222,940 research outputs across all sources so far. This one is in the 49th percentile – i.e., 49% of other outputs scored the same or lower than it.
So far Altmetric has tracked 4,482 research outputs from this source. They receive a mean Attention Score of 3.9. This one has gotten more attention than average, scoring higher than 71% of its peers.
Older research outputs will score higher simply because they've had more time to accumulate mentions. To account for age we can compare this Altmetric Attention Score to the 270,334 tracked outputs that were published within six weeks on either side of this one in any source. This one has gotten more attention than average, scoring higher than 66% of its contemporaries.
We're also able to compare this research output to 76 others from the same source and published within six weeks on either side of this one. This one has gotten more attention than average, scoring higher than 72% of its contemporaries.