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Loss of Cannabinoid Receptor CB1 Induces Preterm Birth

Overview of attention for article published in PLOS ONE, October 2008
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Title
Loss of Cannabinoid Receptor CB1 Induces Preterm Birth
Published in
PLOS ONE, October 2008
DOI 10.1371/journal.pone.0003320
Pubmed ID
Authors

Haibin Wang, Huirong Xie, Sudhansu K. Dey

Abstract

Preterm birth accounting approximate 10% of pregnancies in women is a tremendous social, clinical and economic burden. However, its underlying causes remain largely unknown. Emerging evidence suggests that endocannabinoid signaling via cannabinoid receptor CB1 play critical roles in multiple early pregnancy events in both animals and humans. Since our previous studies demonstrated that loss of CB1 defers the normal implantation window in mice, we surmised that CB1 deficiency would influence parturition events.

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X Demographics

The data shown below were collected from the profile of 1 X user who shared this research output. Click here to find out more about how the information was compiled.
Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 65 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
United States 2 3%
United Kingdom 1 2%
Japan 1 2%
Unknown 61 94%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 13 20%
Student > Master 10 15%
Researcher 9 14%
Student > Bachelor 7 11%
Student > Doctoral Student 5 8%
Other 11 17%
Unknown 10 15%
Readers by discipline Count As %
Agricultural and Biological Sciences 20 31%
Biochemistry, Genetics and Molecular Biology 12 18%
Medicine and Dentistry 11 17%
Pharmacology, Toxicology and Pharmaceutical Science 2 3%
Immunology and Microbiology 2 3%
Other 6 9%
Unknown 12 18%
Attention Score in Context

Attention Score in Context

This research output has an Altmetric Attention Score of 2. This is our high-level measure of the quality and quantity of online attention that it has received. This Attention Score, as well as the ranking and number of research outputs shown below, was calculated when the research output was last mentioned on 06 November 2014.
All research outputs
#16,097,484
of 24,493,651 outputs
Outputs from PLOS ONE
#140,410
of 211,511 outputs
Outputs of similar age
#80,331
of 94,355 outputs
Outputs of similar age from PLOS ONE
#345
of 413 outputs
Altmetric has tracked 24,493,651 research outputs across all sources so far. This one is in the 32nd percentile – i.e., 32% of other outputs scored the same or lower than it.
So far Altmetric has tracked 211,511 research outputs from this source. They typically receive a lot more attention than average, with a mean Attention Score of 15.6. This one is in the 30th percentile – i.e., 30% of its peers scored the same or lower than it.
Older research outputs will score higher simply because they've had more time to accumulate mentions. To account for age we can compare this Altmetric Attention Score to the 94,355 tracked outputs that were published within six weeks on either side of this one in any source. This one is in the 14th percentile – i.e., 14% of its contemporaries scored the same or lower than it.
We're also able to compare this research output to 413 others from the same source and published within six weeks on either side of this one. This one is in the 16th percentile – i.e., 16% of its contemporaries scored the same or lower than it.