@APSPublications @ETTheKidneyguy @LamaQusairi @RickGrimmPhD @AJPRenal This recent paper is a nice complement to ours, raising the possibility that parallel activation of pendrin and KCC3 facilitate electroneutral potassium-bicarbonate excretion, ideally su
KCC3a, a Strong Candidate Pathway for K+ Loss in Alkalemia https://t.co/p9YQXd0814
New Research: KCC3a, a Strong Candidate Pathway for K+ Loss in Alkalemia: Loss-of-function mutations in the human potassium chloride cotransporter-3 (KCC3) cause a hereditary motor sensory neuropathy associated with agenesis of the corpus… https://t.co/NRv
RT @PAWellingMD: A new study by Ferdaus et al in the Delpire group identifies KCC3a as a strong candidate for the long sought-after electro…