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PINK1 Defect Causes Mitochondrial Dysfunction, Proteasomal Deficit and α-Synuclein Aggregation in Cell Culture Models of Parkinson's Disease

Overview of attention for article published in PLOS ONE, February 2009
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Citations

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135 Mendeley
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Title
PINK1 Defect Causes Mitochondrial Dysfunction, Proteasomal Deficit and α-Synuclein Aggregation in Cell Culture Models of Parkinson's Disease
Published in
PLOS ONE, February 2009
DOI 10.1371/journal.pone.0004597
Pubmed ID
Authors

Wencheng Liu, Cristofol Vives-Bauza, Rebeca Acín-Peréz-, Ai Yamamoto, Yingcai Tan, Yanping Li, Jordi Magrané, Mihaela A. Stavarache, Sebastian Shaffer, Simon Chang, Michael G. Kaplitt, Xin-Yun Huang, M. Flint Beal, Giovanni Manfredi, Chenjian Li

Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 135 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
United Kingdom 1 <1%
United States 1 <1%
Czechia 1 <1%
Germany 1 <1%
Unknown 131 97%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 27 20%
Researcher 21 16%
Student > Master 18 13%
Student > Bachelor 13 10%
Other 7 5%
Other 27 20%
Unknown 22 16%
Readers by discipline Count As %
Agricultural and Biological Sciences 46 34%
Neuroscience 21 16%
Biochemistry, Genetics and Molecular Biology 19 14%
Medicine and Dentistry 7 5%
Pharmacology, Toxicology and Pharmaceutical Science 5 4%
Other 8 6%
Unknown 29 21%