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Loss of Raf-1 kinase inhibitor protein (RKIP) is strongly associated with high-grade tumor budding and correlates with an aggressive phenotype in pancreatic ductal adenocarcinoma (PDAC)

Overview of attention for article published in Journal of Translational Medicine, January 2013
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Mentioned by

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1 tweeter

Citations

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11 Dimensions

Readers on

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15 Mendeley
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Title
Loss of Raf-1 kinase inhibitor protein (RKIP) is strongly associated with high-grade tumor budding and correlates with an aggressive phenotype in pancreatic ductal adenocarcinoma (PDAC)
Published in
Journal of Translational Medicine, January 2013
DOI 10.1186/1479-5876-11-311
Pubmed ID
Authors

Eva Karamitopoulou, Inti Zlobec, Beat Gloor, Agathi Kondi-Pafiti, Alessandro Lugli, Aurel Perren

Abstract

Raf-1 kinase inhibitor protein (RKIP) has emerged as a significant metastatic suppressor in a variety of human cancers and is known to inhibit Ras/Raf/MEK/ERK signaling. By suppressing the activation of the NFkB/SNAIL circuit, RKIP can regulate the induction of epithelial-mesenchymal transition (EMT). The aim of this study was to evaluate RKIP expression and to determine its association with clinicopathological features, including EMT in form of tumor budding in pancreatic ductal adenocarcinoma (PDAC).

Twitter Demographics

The data shown below were collected from the profile of 1 tweeter who shared this research output. Click here to find out more about how the information was compiled.

Mendeley readers

The data shown below were compiled from readership statistics for 15 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Unknown 15 100%

Demographic breakdown

Readers by professional status Count As %
Student > Doctoral Student 3 20%
Student > Ph. D. Student 3 20%
Student > Master 3 20%
Other 2 13%
Student > Postgraduate 1 7%
Other 3 20%
Readers by discipline Count As %
Medicine and Dentistry 10 67%
Biochemistry, Genetics and Molecular Biology 3 20%
Psychology 1 7%
Unspecified 1 7%

Attention Score in Context

This research output has an Altmetric Attention Score of 1. This is our high-level measure of the quality and quantity of online attention that it has received. This Attention Score, as well as the ranking and number of research outputs shown below, was calculated when the research output was last mentioned on 14 December 2013.
All research outputs
#2,301,725
of 4,507,509 outputs
Outputs from Journal of Translational Medicine
#519
of 1,265 outputs
Outputs of similar age
#58,977
of 123,236 outputs
Outputs of similar age from Journal of Translational Medicine
#29
of 74 outputs
Altmetric has tracked 4,507,509 research outputs across all sources so far. This one is in the 35th percentile – i.e., 35% of other outputs scored the same or lower than it.
So far Altmetric has tracked 1,265 research outputs from this source. They receive a mean Attention Score of 2.7. This one is in the 43rd percentile – i.e., 43% of its peers scored the same or lower than it.
Older research outputs will score higher simply because they've had more time to accumulate mentions. To account for age we can compare this Altmetric Attention Score to the 123,236 tracked outputs that were published within six weeks on either side of this one in any source. This one is in the 38th percentile – i.e., 38% of its contemporaries scored the same or lower than it.
We're also able to compare this research output to 74 others from the same source and published within six weeks on either side of this one. This one is in the 25th percentile – i.e., 25% of its contemporaries scored the same or lower than it.